Description
Selank (Research Compound)
Tagline: Neuroactive Peptide Analog
Product Description
Selank is a synthetic peptide analog of the endogenous tetrapeptide tuftsin, developed for neuroscience and immunology research. This research compound is widely studied for its potential role in regulating neurotransmitters, modulating immune response, and influencing stress resilience.
Designed for laboratory and scientific research only, Selank provides researchers with a stable, high-purity tool to investigate peptide activity in controlled experimental models.
Why Researchers Choose Selank
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High purity ensures reliable results in controlled experiments.
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Stable formulation suitable for both short- and long-term laboratory studies.
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Ease of handling with clear solubility and reconstitution guidelines.
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Research relevance in neurobiology, stress regulation, and immune modulation.
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Consistent quality control for reproducibility across trials.
Research Data & Handling Tips
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Reconstitution: Selank may be reconstituted in sterile water or appropriate buffer.
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Solubility: Soluble in water; ensure complete dissolution before use.
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Storage: Store lyophilized powder at –20°C; once reconstituted, refrigerate at 2–8°C.
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Stability: Stable for up to 24 months (lyophilized). Use within 4–8 weeks after reconstitution.
Important Note
For laboratory and scientific research only. Not for human consumption.
Research
Research
Neurological Function & Cognition
Studies suggest Selank modulates neurotransmitter activity, particularly within the serotonin and dopamine systems, which may influence learning, memory, and attention processes [1].
Stress & Anxiety Pathways
Preclinical findings indicate that Selank may help regulate the hypothalamic-pituitary-adrenal (HPA) axis, offering insights into stress-related responses [2].
Immune Modulation
Research shows Selank derivatives may affect cytokine balance, linking neuropeptide activity with immune system regulation [3].
Metabolic Regulation
Preliminary studies suggest possible roles in energy homeostasis and neuroendocrine balance, making Selank relevant to broader systemic research [4].
References
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Semenova TP, Kozlovskaya MM, Zakharova NM, Zuikov AV. Effect of Selank on cognitive processes after damage inflicted to the cerebral catecholamine system during early ontogeny. Bull Exp Biol Med. 2007;144:689-691.
https://link.springer.com/article/10.1007/s10517-007-0406-2
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Volkova A, et al. Selank administration affects the expression of some genes involved in neurotransmission in the frontal cortex of rats. Front Pharmacol. 2016;7:31.
https://www.frontiersin.org/journals/pharmacology/articles/10.3389/fphar.2016.00031/full
and (stress model): Mjasoedov N, et al. Selank peptide causes changes in gene expression in the rat brain after acute restraint stress (RNA-Seq study). Biology Bulletin Reviews. 2024.
https://link.springer.com/article/10.1134/S2635167624601335
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Kolomin T, et al. Expression of inflammation-related genes in mouse spleen under tuftsin analogue Selank action. Regul Pept. 2011;170:18-23.
https://pillbuys.com/research/Selank/19.pdf ScienceDirect
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Myasoedov NF, et al. Effect of Selank on functional state of rat hepatocytes under conditions of acute restraint stress. Bull Exp Biol Med. 2017;163:715-719.https://www.sciencedirect.com/science/article/pii/S0167011511000863?utm_source=chatgpt.com
https://link.springer.com/article/10.1007/s10517-017-3817-8
